Author

E. Alvirez

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:whole-genome sequencing· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:statistical model· 1

Frequent Co-Authors

E. Gharzouzi· 2 shared H. Lou· 2 shared J. F. Boland· 2 shared L. Mirabello· 2 shared M. Dean· 2 shared M. Yeager· 2 shared A. Albanez· 1 shared D. Roberson· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersCancer Biology

E. Gharzouzi

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2 papers

Recent·Most cited

Extrachromosomal Amplification of Human Papillomavirus Episomes as a Mechanism of Cervical Carcinogenesis

N. M. Rossi, J. Dai, Y. Xie, H. Lou, J. F. Boland, M. Yeager, R. Orozco, E. Alvirez, L. Mirabello, E. Gharzouzi, M. Dean

The paper identifies extrachromosomal HPV16 episomes as a mechanism of cervical carcinogenesis, showing that they can cause cancer without integration through rearranged and multimer episomes.

bioRxiv · Oct 22, 2021 · PreprintCancer Biology

Human papillomavirus 16 positive cervical cancer in Guatemala: The D2 and D3 sublineages differ in integration rate and age of diagnosis

H. Lou, J. F. Boland, E. Torres-Gonzalez, A. Albanez, W. Zhou, M. Steinberg, L. Diaw, J. Mitchell, D. Roberson, M. Cullen, L. Garland, S. Bass, R. Burk, M. Yeager, N. Wentzensen, , M. Dean

This study compares HPV16 D2 and D3 sublineages in Guatemalan cervical cancer samples, finding differences in integration rates, age of diagnosis, and somatic mutation frequency.

bioRxiv · Jan 7, 2020Cancer Biology