Author

E. Ferraro

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Ecology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:comparative / phylogenetic analysis· 1 Research Focus:differential expression· 1 Research Focus:ecology & biodiversity· 1 Assay:field survey / ecological data· 1

Frequent Co-Authors

A. Brugnoli· 1 shared A. Dornburg· 1 shared A. Sintou· 1 shared A. W. Thompson· 1 shared C. Mansfield· 1 shared D. J. Wcisel· 1 shared F. Cagnacci· 1 shared F. Ossi· 1 shared

Activity by Year

2019-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperEcology

A. Brugnoli

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Holosteans contextualize the role of the teleost genome duplication in promoting the rise of evolutionary novelties in the ray-finned fish innate immune system

A. Dornburg, D. J. Wcisel, K. Zapfe, E. Ferraro, L. Roupe-Abrams, A. W. Thompson, I. Braasch, T. Ota, J. A. Yoder

This study investigates the innate immune receptor diversity in Holostei, showing that their gene families predate the teleost genome duplication and rival teleost diversity, challenging assumptions about the TGD's role in immune innovation.

bioRxiv · Jun 11, 2021Immunology

Preference and familiarity mediate spatial responses of a large herbivore to experimental manipulation of resource availability

N. Ranc, P. R. Moorcroft, K. W. Hansen, F. Ossi, T. Sforna, A. Brugnoli, E. Ferraro, F. Cagnacci

This study shows that roe deer adjust their movement and home range size based on resource availability and individual preference, highlighting the role of site fidelity in home range maintenance.

bioRxiv · Dec 12, 2019Ecology

Persistent anti-heart autoimmunity causes cardiomyocyte damage in chronic heart failure.

A. Sintou, S. el Rifai, C. Mansfield, S. Rothery, J. L. Sanchez Alonso, S. Narodden, K. Sharma, E. Ferraro, M. G. Hasham, P. Swiatlowska, S. E. Harding, N. Rosenthal, J. Gorelik, S. Sattler

This study shows that persistent anti-heart autoimmunity contributes to cardiomyocyte damage and chronic heart failure progression in rats after myocardial infarction.

bioRxiv · Mar 13, 2019Immunology