Author

N. Rosenthal

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1 Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Research Focus:cell type annotation· 4 Research Focus:differential expression· 4 Organism:mouse· 4 Research Focus:host-pathogen & immune response· 3 Assay:imaging· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

S. E. Harding· 3 shared S. Sattler· 3 shared E. Forte· 2 shared J. Branca· 2 shared M. G. Hasham· 2 shared O. Bedard· 2 shared A. Chiramel· 1 shared A. Lota· 1 shared

Activity by Year

2017-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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3 shared papersImmunology

S. E. Harding

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Genetically diverse mouse models of SARS-CoV-2 infection reproduce clinical variation in type I interferon and cytokine responses in COVID-19

S. Robertson, O. Bedard, K. McNally, C. Shaia, C. Clancy, M. Lewis, R. Broeckel, A. Chiramel, J. G. Shannon, G. Sturdevant, R. Rosenke, S. L. Anzick, E. Forte, C. Preuss, C. Baker, , S. Best

The paper uses genetically diverse mice to model varied innate immune responses to SARS-CoV-2 infection, showing differences in survival, viral replication, and cytokine profiles.

bioRxiv · Sep 17, 2021Microbiology

Myocardial damage induced by a single high dose of isoproterenol in C57BL/6J mice triggers a persistent adaptive immune response against the heart.

E. Forte, M. Panahi, F. S. Ng, J. J. Boyle, J. Branca, O. Bedard, M. G. Hasham, L. Benson, S. E. Harding, N. Rosenthal, S. Sattler

This study shows that a single high dose of isoproterenol in mice causes myocardial damage and a persistent adaptive immune response against the heart, making it a suitable model for studying anti-heart autoimmunity.

bioRxiv · Feb 27, 2020Immunology

Persistent anti-heart autoimmunity causes cardiomyocyte damage in chronic heart failure.

A. Sintou, S. el Rifai, C. Mansfield, S. Rothery, J. L. Sanchez Alonso, S. Narodden, K. Sharma, E. Ferraro, M. G. Hasham, P. Swiatlowska, S. E. Harding, N. Rosenthal, J. Gorelik, S. Sattler

This study shows that persistent anti-heart autoimmunity contributes to cardiomyocyte damage and chronic heart failure progression in rats after myocardial infarction.

bioRxiv · Mar 13, 2019Immunology

Detection of acute TLR-7 agonist-induced hemorrhagic myocarditis in mice by refined multi-parametric quantitative cardiac MRI.

N. Baxan, A. Papanikolaou, I. Salles-Crawley, A. Lota, R. Chowdhury, O. Dubois, J. Branca, M. Hasham, N. Rosenthal, S. Prasad, L. Zhao, S. E. Harding, S. Sattler

This study identifies hemorrhagic myocarditis in mice induced by TLR-7 agonist using multi-parametric cardiac MRI, showing distinct T1, T2, and T2* relaxation patterns.

bioRxiv · Dec 21, 2018Physiology

Single cell transcriptional profiling reveals cellular diversity, communication, and sexual dimorphism in the mouse heart

D. A. Skelly, G. T. Squiers, M. A. McLellan, M. T. Bolisetty, P. Robson, N. Rosenthal, A. R. Pinto

This study uses scRNA-seq to analyze the mouse heart's non-myocyte cells, revealing cellular diversity, communication networks, and sexual dimorphism in gene expression.

bioRxiv · Oct 12, 2017Cell Biology