Author

E. Peris

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

B. Chanclon· 1 shared C. Jonsson· 1 shared C. S. Olofsson· 1 shared E. Banke· 1 shared I. W. Asterholm· 1 shared M. Bauza-Thorbrugge· 1 shared P. Rorsman· 1 shared P. Stralfors· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPhysiology

B. Chanclon

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Adipocyte-specific ablation of the Ca2+ pump SERCA2 impairs whole-body metabolic function and reveals the diverse metabolic flexibility of white and brown adipose tissue.

M. Bauza-Thorbrugge, E. Banke, B. Chanclon, E. Peris, Y. Wu, S. Musovic, C. Jonsson, P. Stralfors, P. Rorsman, C. S. Olofsson, I. W. Asterholm

This study shows that reducing SERCA2 in adipocytes impairs metabolic function, with white adipose tissue showing increased glucose uptake and brown adipose tissue showing reduced mitochondrial function.

bioRxiv · Mar 11, 2022Physiology