Author

G. Schena

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Physiology· 1

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Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

H. Shen· 1 shared K. Dong· 1 shared L. Onuchic· 1 shared M. J. Caplan· 1 shared N. P. Gresko· 1 shared S. Somlo· 1 shared V. Padovano· 1 shared V. Rajendran· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperPhysiology

H. Shen

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1 paper

Recent·Most cited

The C-terminal tail of polycystin-1 suppresses cystic disease in a mitochondrial enzyme-dependent fashion

L. Onuchic, V. Padovano, G. Schena, V. Rajendran, K. Dong, N. P. Gresko, X. Shi, H. Shen, S. Somlo, M. J. Caplan

The study shows that a C-terminal fragment of PC1 suppresses cystic disease in a murine model by interacting with a mitochondrial enzyme, affecting metabolism and mitochondrial function.

bioRxiv · Dec 21, 2021 · PreprintPhysiology