Author

I. Braren

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

G. Mearini· 2 shared L. Carrier· 2 shared N. Pietsch· 2 shared S. Schlossarek· 2 shared A. Madsen· 1 shared A. T. Zech· 1 shared B. Geertz· 1 shared B. L. Prosser· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersPhysiology

G. Mearini

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Chronic activation of tubulin tyrosination in HCM mice and human iPSC-engineered heart tissues improves heart function

N. Pietsch, C. Y. Chen, S. Kupsch, L. Bacmeister, B. Geertz, M. Herera-Rivero, H. Voss, E. Krämer, I. Braren, D. Westermann, H. Schlüter, G. Mearini, S. Schlossarek, J. van der Velden, M. A. Caporizzo, , L. Carrier

bioRxiv · May 25, 2023Physiology

ACTN2 mutant causes proteopathy in human iPSC-derived cardiomyocytes

A. T. Zech, M. Prondzynski, S. R. Singh, N. Pietsch, E. Orthey, E. Alizoti, J. Busch, A. Madsen, C. S. Behrens, M. Meyer-Jens, G. Mearini, M. D. Lemoine, E. Kraemer, D. Mosqueira, S. Virdi, , L. Carrier

This study shows that an ACTN2 mutation leads to proteopathy in human iPSC-derived cardiomyocytes through protein aggregation and activation of proteolytic systems.

bioRxiv · Oct 28, 2021Cell Biology