Author

I. Gavrish

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Assay:imaging· 1 Approach:microscopy & imaging analysis· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

I. Kramnik· 2 shared A. Belkina· 1 shared A. E. Tseng· 1 shared A. K. O'Connel· 1 shared C. E. Thurman· 1 shared D. N. Kotton· 1 shared H. Kiyokawa· 1 shared H. P. Gertje· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersImmunology

I. Kramnik

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Aberrant macrophage activation and maladaptive lung repair promote tuberculosis progression uniquely in the lung

S. M. Yabaji, S. Lata, A. E. Tseng, P. B. Araveti, M. Lo, I. Gavrish, A. K. O'Connel, H. P. Gertje, A. Belkina, C. E. Thurman, H. Kiyokawa, D. N. Kotton, S. Tan, J. J. Endsley, W. R. Bishai, , I. Kramnik

This study identifies aberrant macrophage activation and maladaptive lung repair as key factors promoting tuberculosis progression in the lung using a novel mouse model.

bioRxiv · Oct 17, 2023 · PreprintImmunology

Dissemination and progression of pulmonary Mycobacterium avium infection in mouse model are associated with type 2 macrophage activation

R. Rosenbloom, I. Gavrish, K. Seidel, I. Kramnik, N. A. Crossland

This study shows that B6.Sst1S mice develop more severe pulmonary M. avium infection with increased type 2 macrophage activation and reduced T cell density compared to wild-type mice.

bioRxiv · Jun 23, 2021 · PreprintPathology