Author

J. Cendrowski

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Approach:experimental assay· 3 Organism:human· 3 Research Focus:development & morphogenesis· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

M. Miaczynska· 3 shared M. Mazur· 2 shared M. Wrobel· 2 shared A. Dobrzyn· 1 shared A. Dziewulska· 1 shared A. Kominek· 1 shared A. Paziewska· 1 shared A. Zeigerer· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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3 shared papersCell Biology

M. Miaczynska

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Author profile →

3 papers

Recent·Most cited

NF-kappaB and JNK pathways mediate metabolic adaptation upon ESCRT-I deficiency

J. Cendrowski, M. Wrobel, M. Mazur, B. Jary, S. Wang, M. Korostynski, A. Dziewulska, M. Rohm, A. Dobrzyn, A. Zeigerer, M. Miaczynska

This study shows that ESCRT-I deficiency leads to metabolic reprogramming via NF-kappaB and JNK pathways, causing increased glycolysis and lipid accumulation.

bioRxiv · Jun 12, 2024Cell Biology

ESCRT-I controls lysosomal membrane protein homeostasis and restricts MCOLN1-dependent TFEB/TFE3 signaling.

M. Wrobel, E. Szymanska, N. Budick-Harmelin, K. Kolmus, K. Goryca, M. Dabrowska, A. Paziewska, M. Mikula, J. Cendrowski, M. Miaczynska

This study shows ESCRT-I regulates lysosomal membrane protein homeostasis and modulates TFEB/TFE3 signaling through MCOLN1-dependent Ca2+ channels.

bioRxiv · Jul 26, 2021 · PreprintCell Biology

Splicing variation of BMP2K balances endocytosis, COPII trafficking and autophagy in erythroid cells

J. Cendrowski, M. Kaczmarek, K. Kuzmicz-Kowalska, M. Mazur, K. Jastrzebski, M. Brewinska-Olchowik, A. Kominek, K. Piwocka, M. Miaczynska

The paper identifies splicing variants of BMP2K that regulate erythroid differentiation by modulating COPII trafficking and autophagy through interactions with SEC16A.

bioRxiv · May 5, 2020 · PreprintCell Biology