Author

J. Ceron

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Cell Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:C. elegans· 4 Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:drug response / drug discovery· 2 Assay:behavioral assay· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

C. Martinez-Fernandez· 3 shared A. Villanueva· 2 shared D. Brena· 2 shared E. Nadal· 2 shared S. Honnen· 2 shared A. Esteve-Codina· 1 shared A. Miranda-Vizuete· 1 shared C. I. Holmberg· 1 shared

Activity by Year

2017-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCell Biology

C. Martinez-Fernandez

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

AKIR-1 Regulates Proteasome Localization and Function in Caenorhabditis elegans

J. Pispa, E. Mikkonen, L. Arpalahti, C. Jin, C. Martinez-Fernandez, J. Ceron, C. I. Holmberg

This study identifies AKIR-1 as a regulator of proteasome localization and function in C. elegans, showing its role in nuclear proteasome transport and proteostasis.

bioRxiv · Oct 3, 2022 · PreprintCell Biology

Insights into cisplatin-induced neurotoxicity and mitochondrial dysfunction in Caenorhabditis elegans

C. Martinez-Fernandez, M. Bergamino, D. Brena, N. Ventura, S. Honnen, A. Villanueva, E. Nadal, J. Ceron

This study investigates cisplatin-induced neurotoxicity and mitochondrial dysfunction in C. elegans using genetic, biochemical, and behavioral assays.

bioRxiv · Jun 3, 2021Cell Biology

CRISPR editing of sftb-1/SF3B1 in C. elegans allows the identification of synthetic interactions with cancer-related mutations and the chemical inhibition of splicing

X. Serrat, D. Kukhtar, E. Cornes, A. Esteve-Codina, H. Benlloch, G. Cecere, J. Ceron

The paper uses CRISPR to edit sftb-1/SF3B1 in C. elegans to identify synthetic interactions with cancer mutations and test splicing inhibitors.

bioRxiv · Jun 11, 2019 · PreprintCancer Biology

Genetic and cellular sensitivity of Caenorhabditis elegans to the chemotherapeutic agent cisplatin

F. J. Garcia-Rodriguez, C. Martinez-Fernandez, D. Brena, E. Nadal, M. Boxem, S. Honnen, A. Miranda-Vizuete, A. Villanueva, J. Ceron

This study investigates cisplatin sensitivity in C. elegans, identifying factors like DAF-16/FOXO, SKN-1/Nrf2, and the intrinsic apoptotic pathway, and discovers a resistance mechanism involving trxr-1 and selenocysteine.

bioRxiv · Dec 12, 2017Cancer Biology