Author

J. Lallement

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

A. Marino· 1 shared C. Amouyal· 1 shared C. Beauloye· 1 shared C. Cruciani-Guglielmacci· 1 shared C. Dessy· 1 shared C. Magnan· 1 shared D. Rainteau· 1 shared E. P. Daskalopoulos· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperPhysiology

A. Marino

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2 papers

Recent·Most cited

When the liver is in poor condition, so is the heart - cardiac remodelling in MASH mouse models

S. Bott, J. Lallement, A. Marino, E. P. Daskalopoulos, C. Beauloye, H. Esfahani, C. Dessy, I. Leclercq

This study examines cardiac remodelling in MASH mouse models, finding adverse structural changes in the heart linked to liver disease, not overfeeding or aging.

bioRxiv · May 1, 2024Physiology

Hepatocyte serine palmitoyl transferase 2 deficiency promotes liver C16:0-ceramide accumulation through sphingomyelin hydrolysis and leads to liver damage and dysfunction in mice

J. Lallement, I. Raho, G. Merlen, D. Rainteau, M. Croyal, M. Schiffano, N. Kassis, I. Doignon, M. Soty, M. Krempf, C. Amouyal, H. Le Stunff, C. Magnan, T. Tordjmann, C. Cruciani-Guglielmacci

This study shows that Sptlc2 deficiency in hepatocytes increases liver ceramide levels via sphingomyelin hydrolysis, leading to liver damage, dysfunction, and metabolic changes in mice.

bioRxiv · Sep 16, 2021 · PreprintPhysiology