Author

M. Croyal

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Physiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Assay:metabolomics· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

A. Ayer· 1 shared B. Cariou· 1 shared C. Amouyal· 1 shared C. Cruciani-Guglielmacci· 1 shared C. Gratas· 1 shared C. Le May· 1 shared C. Magnan· 1 shared C. Pecqueur· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPhysiology

A. Ayer

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

PCSK9 inhibition protects mice from food allergy

V. Lorant, M. Klein, D. Garcon, T. Sotin, S. Frey, M.-A. Cheminant, A. Ayer, M. Croyal, L. Flet, L. Colas, B. Cariou, G. Bouchaud, C. Le May

This study shows that PCSK9 inhibition protects mice from gliadin-induced food allergy symptoms, independent of LDLR.

bioRxiv · Oct 10, 2023Physiology

Pyruvate carboxylation identifies Glioblastoma Stem-like Cells opening new metabolic strategy to prevent tumor recurrence

O. Renoult, C. Gratas, N. Joalland, M. Laurent--Blond, H. Awada, M. Bourgeois, L. Oliver, S. Chiavassa, M. Croyal, F. Paris, C. Pecqueur

The paper identifies Glioblastoma Stem-like Cells (GSCs) through unique metabolic features and suggests targeting pyruvate carboxylation to prevent tumor recurrence.

bioRxiv · Jul 18, 2022Cancer Biology

Hepatocyte serine palmitoyl transferase 2 deficiency promotes liver C16:0-ceramide accumulation through sphingomyelin hydrolysis and leads to liver damage and dysfunction in mice

J. Lallement, I. Raho, G. Merlen, D. Rainteau, M. Croyal, M. Schiffano, N. Kassis, I. Doignon, M. Soty, M. Krempf, C. Amouyal, H. Le Stunff, C. Magnan, T. Tordjmann, C. Cruciani-Guglielmacci

This study shows that Sptlc2 deficiency in hepatocytes increases liver ceramide levels via sphingomyelin hydrolysis, leading to liver damage, dysfunction, and metabolic changes in mice.

bioRxiv · Sep 16, 2021 · PreprintPhysiology