Author

J. Maynard

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Assay:DNA methylation· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. L. Burlingame· 1 shared A. M. De Marzo· 1 shared A. Meeker· 1 shared B. Ozbek· 1 shared C. Heaphy· 1 shared E. Shrestha· 1 shared H. Zeng· 1 shared H.-B. Ruan· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

A. L. Burlingame

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2 papers

Recent·Most cited

O-GlcNAc glycosylation orchestrates fate decision and niche function of bone marrow stromal progenitors

Z. Zhang, Z. Huang, M. Awad, M. Elsalanty, J. Cray, L. E. Ball, J. Maynard, A. L. Burlingame, H. Zeng, K. C. Mansky, H.-B. Ruan

This study identifies O-GlcNAc glycosylation as a regulator of bone marrow stromal progenitor fate and niche function, influencing osteogenic/adipogenic differentiation and hematopoiesis.

bioRxiv · Dec 21, 2022Physiology

Oncogenic gene fusions in non-neoplastic precursors as evidence that bacterial infection initiates prostate cancer

E. Shrestha, J. Coulter, W. Guzman, B. Ozbek, L. Mummert, S. Ernst, J. Maynard, A. Meeker, C. Heaphy, M. Haffner, A. M. De Marzo, K. S. Sfanos

This study shows bacterial infections initiate prostate cancer by causing DNA breaks that lead to oncogenic TMPRSS2:ERG fusions in precancerous lesions like PIA.

bioRxiv · Jul 27, 2020Cancer Biology