Author

Jianwei Li

Shantou UniversityPrimary ORCID profile (0000-0002-9795-2635 · 1 paper)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Jianwei LiiD1 paper · Hebei University of Technology

Biology Disciplines

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Cell Biology· 1 Neuroscience· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Organism:human· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Baihong LiiD· 1 shared Bing Yuan HuangiD· 1 shared Bingying Lin· 1 shared Erxiang Wang· 1 shared Fei FangiD· 1 shared Fen WangiD· 1 shared Feng LiuiD· 1 shared Feng‐Lai YuaniD· 1 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperPathology

Baihong LiORCID

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3 papers

Recent·Most cited

Genome-wide consensus transcriptional signatures identify synaptic pruning linking Alzheimer's disease and epilepsy

Huihong Li, Zhiran Xie, Yuxuan Tian, Ruoyin Zhou, Yaxi Yang, Bingying Lin, Si Chen, Jie Wu, Zihan Deng, Jianwei Li, Mingjie Chen, Xueke Liu, Yushan Sun, Naili Wei, Bing Yuan Huang, Xiaoyu Ji

The paper identifies a conserved microglial synaptic pruning pathway linked to Alzheimer's disease and epilepsy through genome-wide transcriptomic analysis across multiple models and human cohorts.

Molecular Psychiatry · Oct 26, 2025Peer-reviewedNeuroscience

O-GlcNAcylation-Ubiquitin Crosstalk of METTL1 Drives m7G Epitranscriptomic Collapse and Lipid Metabolic Reprogramming in Diabetic Cardiomyopathy

Xin Gu, Haoyu Meng, Jiabao Liu, Yan Liang, Baihong Li, Fen Wang, Qian S. Liu, Zhixuan Zhang, Jialong Liang, Xiaodong Zhang, Jialong Sun, Jianwei Li, Feng Liu, Xiao Wang, Guangyi Huang, , Yingqiang Du

This study identifies METTL1 O-GlcNAcylation and ubiquitination as key regulators of m7G modification and lipid metabolism in diabetic cardiomyopathy, with a drug targeting this pathway showing therapeutic potential.

bioRxiv · Jul 7, 2025 · PreprintPathology

Matrix Vesicles from Osteoblasts Promote Atherosclerotic Calcification

Xiaoli Wang, Jie Ren, Fei Fang, Erxiang Wang, Jianwei Li, Weihong He, Zhen Zhang, Yang Shen, Xiaoheng Liu

This study shows osteoblast-derived matrix vesicles (Ost-MVs) contribute to atherosclerotic calcification by being released into circulation and transported to plaques, where they exacerbate calcification through the Ras-Raf-ERK pathway.

bioRxiv · Apr 18, 2024Cell Biology