Author

K. Kirschbaum

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 1

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Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

A. Debes· 1 shared A. M. Zeiher· 1 shared B. Schuhmacher· 1 shared C. Muller-Tidow· 1 shared D. John· 1 shared E. Nagel· 1 shared F. Leuschner· 1 shared G. Luxan· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

A. Debes

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1 paper

Recent·Most cited

DNMT3A clonal hematopoiesis-driver mutations induce cardiac fibrosis by paracrine activation of fibroblasts

M. Shumliakivska, G. Luxan, I. Hemmerling, W. T. Abplanalp, X. Li, M. Scheller, C. Muller-Tidow, F. Leuschner, B. Schuhmacher, A. Debes, S.-F. Glaser, M. Muhly Reinholz, K. Kirschbaum, J. Hoffmann, E. Nagel, , S. Dimmeler

This study shows that DNMT3A mutations in hematopoietic cells drive cardiac fibrosis by activating fibroblasts through paracrine signaling, contributing to heart failure progression.

bioRxiv · Jan 7, 2023Physiology