Author

K. Woroniecka

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

E. C. Lerner· 2 shared P. E. Fecci· 2 shared A. M. Hoyt-Miggelbrink· 1 shared A. Miggelbrink· 1 shared A. Mohan· 1 shared D. S. Wilkinson· 1 shared D. Wilkinson· 1 shared J. B. Finlay· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersImmunology

E. C. Lerner

Mutual co-authors in cluster

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintAug 11, 2023

Antigen presentation by tumor-associated macrophages drives tumor-infiltrating T cells from a progenitor exhaustion state to terminal exhaustion

J. Waibl Polania, A. M. Hoyt-Miggelbrink, W. H. Tomaszewski…·P. E. Fecci

The paper identifies tumor-associated macrophages as key drivers of T cell exhaustion transition, showing their role in shifting progenitor exhausted T cells to terminal exhaustion and impacting immunotherapy response.

DOI 10.1101/2023.08.11.553007

humanbulk RNA-seq

𝕏 PostBluesky

ImmunologybioRxivPreprintFeb 2, 2023

A Novel MHC-Independent Mechanism of Tumor Cell Killing by CD8+ T Cells

E. C. Lerner, K. Woroniecka, V. D'Anniballe…·P. E. Fecci

This study shows CD8+ T cells can kill MHC-I-deficient tumor cells via NKG2D-NKG2DL interactions, independent of antigen recognition, and identifies this pathway as a potential therapeutic target.

DOI 10.1101/2023.02.02.526713

mousehuman

𝕏 PostBluesky