K. Yoshikawa

This study shows that NFYAv2 promotes anti-tumor effects in HCC by upregulating PCK1, leading to increased ROS and energy crisis, and suggests it as a potential therapeutic target.

The paper shows NFYAv1 promotes TNBC malignancy by enhancing lipid metabolism, and targeting this axis could be a therapeutic strategy.

The paper proposes a genomic mechanism explaining cell-fate change through critical points and attractor dynamics in genome expression.

This paper examines the genomic mechanism determining cell-fate changes from embryo to cancer development using self-organized criticality as a framework.

This study compares the effects of 1-propanol and 2-propanol on DNA structure using fluorescence microscopy and CD measurements, showing different conformational transitions and secondary structure changes.