Author

L. Cords

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:metabolism & bioenergetics· 1 Organism:mouse· 1

Frequent Co-Authors

B. Bodenmiller· 2 shared A. Curioni-Fontecedro· 1 shared A. Soltermann· 1 shared A. V. Sarapulov· 1 shared C. Langwieder· 1 shared E. Kuokkanen· 1 shared I. Opitz· 1 shared L. Grob· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersCancer Biology

B. Bodenmiller

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A Cancer-Associated Fibroblast Classification Framework for Single-Cell Data

L. Cords, S. Tietscher, T. Anzeneder, C. Langwieder, M. Rees, N. de Souza, B. Bodenmiller

The paper classifies cancer-associated fibroblasts (CAFs) using single-cell RNA sequencing data from breast cancer patients and validates the classification in other cancer types.

bioRxiv · Dec 14, 2022 · PreprintCancer Biology

Acquired resistance to anti-PD1 therapy in patients with NSCLC reveals changes in T cell phenotypes and MET amplification

S. Hiltbrunner, L. Cords, S. Kasser, S. N. Freiberger, S. Kreutzer, N. C. Toussaint, L. Grob, I. Opitz, M. Messerli, M. Zoche, A. Soltermann, M. Rechsteiner, M. van den Broek, B. Bodenmiller, A. Curioni-Fontecedro

This study identifies two patterns of acquired resistance to anti-PD1 therapy in NSCLC patients, including reduced T cell infiltration and MET amplification.

bioRxiv · Apr 8, 2022 · PreprintCancer Biology

Missing-In-Metastasis / Metastasis Suppressor 1 modulates B cell receptor signaling, B cell metabolic potential and T cell-independent immune responses.

A. V. Sarapulov, P. Petrov, S. Hernandez-Perez, V. Sustar, E. Kuokkanen, R. Vinod, M. Vainio, L. Cords, M. Fritzsche, Y. Carrasco, P. Mattila

This study shows that MIM/MTSS1 regulates B cell receptor signaling, metabolic activity, and T cell-independent immune responses, with implications for cancer biology.

bioRxiv · Mar 8, 2020Immunology