Author

L. H. Boise

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:deep learning· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. P. Wiita· 1 shared B. G. Barwick· 1 shared C. Driessen· 1 shared C. S. Greene· 1 shared D. B. Weir· 1 shared G. P. Way· 1 shared I. D. Ferguson· 1 shared M. C. Mariano· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. P. Wiita

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cell BiologybioRxivPreprintJan 27, 2021

Caspase3-deficient cells require fibronectin for protection against autophagy-dependent death

D. B. Weir, L. H. Boise

This study shows that fibronectin protects Casp3-deficient cells from autophagy-dependent death during serum withdrawal by preventing cytochrome c release and caspase-independent cell death.

DOI 10.1101/2021.01.27.428460

mouseexperimental assay

𝕏 PostBluesky

Cancer BiologybioRxivFeb 28, 2019

Integrated Phosphoproteomics and Transcriptional Classifiers Reveal Hidden RAS Signaling Dynamics in Multiple Myeloma

Y.-H. T. Lin, G. P. Way, B. G. Barwick…·A. P. Wiita

The paper uses phosphoproteomics and transcriptomics to analyze RAS signaling in multiple myeloma, identifying distinct signaling patterns and clinical outcomes associated with NRAS and KRAS mutations.

DOI 10.1182/bloodadvances.2019000303

humanproteomics

𝕏 PostBluesky