Cancer BiologybioRxivPreprint
Drug resistance mechanisms create targetable proteostatic vulnerabilities in Her2+ breast cancers
K. Komurov, N. Singh, L. Privette-Vinnedge
This study identifies proteostatic vulnerabilities in Her2+ breast cancers during resistance to Her2 inhibitors, showing that targeting ER quality control can overcome resistance.
DOI 10.1101/2021.08.18.456887