Author

M. Bertolini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Assay:flow cytometry· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. B. M. Astrand· 1 shared A. G. Messenger· 1 shared A. Gilhar· 1 shared A. J. McDonagh· 1 shared A. Keren· 1 shared A. Maleki-Dizaji· 1 shared A. Rossi· 1 shared B. Bukau· 1 shared

Activity by Year

2018-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperImmunology

A. B. M. Astrand

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3 papers

Recent·Most cited

Involvement of ILC1-like innate lymphocytes in human autoimmunity, lessons from alopecia areata

R. Laufer Britva, A. Keren, M. Bertolini, Y. Ullmann, R. Paus, A. Gilhar

This study shows that ILC1-like cells can induce alopecia areata ex vivo and in vivo, challenging the view that AA is solely CD8+ T cell-driven.

bioRxiv · Jun 22, 2022Immunology

Translational Activity Controls Ribophagic Flux and Turnover of Distinct Ribosome Pools

J. Trendel, M. Aleksic, M. Bertolini, M. Jochem, G. Kramer, S. Pfeffer, B. Bukau, J. Krijgsveld

The paper investigates ribosome turnover in human cells, showing distinct subunit pools with different turnover rates and demonstrating that translation inhibition increases 80S ribosome pool size and induces ribophagy.

bioRxiv · May 13, 2022 · PreprintBiochemistry

Alopecia areata patients show deficiency of FOXP3+CD39+ T regulatory cells and clonotypic restriction of Treg TCR?-chain, which highlights the immunopathological aspect of the disease

A. B. M. Astrand, F. N. Hamed, M. Bertolini, A. Rossi, A. Maleki-Dizaji, A. G. Messenger, A. J. McDonagh, R. Tazi-Ahnini

The paper examines Treg cell subsets and TCRβ clonotypes in alopecia areata patients, finding reduced FOXP3+CD39+ Tregs and specific clonotypic differences that suggest potential therapeutic targets.

bioRxiv · Dec 22, 2018Immunology