Author

M. Bouche

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cell Biology· 1 Ecology· 1

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Amirshaghaghi· 1 shared A. Bongiovanni· 1 shared A. Coulon· 1 shared A. D'Amico· 1 shared A. E. Tward· 1 shared A. Frey-Roos· 1 shared A. K. Andrianov· 1 shared A. Loison· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperBioengineering

A. Amirshaghaghi

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A biodegradable "one-for-all" nanoparticle for multimodality imaging and enhanced photothermal treatment of breast cancer

J. C. Hsu, D. Barragan, A. E. Tward, M. Hajfathalian, A. Amirshaghaghi, K. J. Mossburg, D. N. Rosario-Berrios, M. Bouche, A. K. Andrianov, E. J. Delikatny, D. P. Cormode

This paper develops AgPCPP, biodegradable nanoparticles that enhance NIR imaging and photothermal therapy for breast cancer, showing safety and efficacy in vivo.

bioRxiv · Nov 28, 2023Bioengineering

Identifying the environmental drivers of corridors and predicting connectivity between seasonal ranges in multiple populations of Alpine ibex (Capra ibex) as tools for conserving migration

V. Chauveau, M. Garel, C. Toïgo, P. Anderwald, M. Beurier, Y. Bunz, M. Bouche, F. Cagnacci, M. Canut, J. Cavailhes, I. Champly, F. Filli, A. Frey-Roos, G. Gressmann, I. Herfindal, , P. Marchand

The paper identifies environmental drivers of migration corridors and predicts connectivity between seasonal ranges of Alpine ibex populations to aid conservation.

bioRxiv · Mar 2, 2023Ecology

Pharmacological tuning of microRNA in FAP-derived Extracellular Vesicles by HDAC inhibitors promotes regeneration and reduces fibrosis in dystrophic muscles.

M. Sandona, S. Consalvi, L. Tucciarone, M. De Bardi, M. Scimeca, D. F. Angelini, V. Buffa, A. D'Amico, E. S. Bertini, S. Cazzaniga, P. Bettica, M. Bouche, A. Bongiovanni, P. L. Puri, V. Saccone

The paper shows that HDAC inhibitors increase microRNA levels in FAP-derived EVs, enhancing muscle regeneration and reducing fibrosis in dystrophic models.

bioRxiv · Feb 15, 2020 · PreprintCell Biology