Author

M. F. Khalid

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Organism:E. coli· 1 Organism:mouse· 1

Frequent Co-Authors

C. L. Pin· 1 shared J. Shi· 1 shared J. Toma· 1 shared M. Hamid· 1 shared M. Martin· 1 shared M. Pasca Di Magliano· 1 shared N. Azizi· 1 shared N. Steele· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

C. L. Pin

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Author profile →

2 papers

Recent·Most cited

The Solvation of the E. coli CheY Phosphorylation SiteMapped by XFMS

M. Hamid, M. F. Khalid, S. U. Chaudhary, S. khan

The paper reanalyzes XFMS data to show polar residues around the phosphorylated CheY site form a solvation network stabilizing the apo-CheY fold, with 18R as a central node.

bioRxiv · Aug 30, 2022Biochemistry

Loss of Activating Transcription Factor 3 prevents KRAS-mediated pancreatic cancer

N. Azizi, J. Toma, M. Martin, M. F. Khalid, N. Steele, J. Shi, M. Pasca Di Magliano, C. L. Pin

This study shows that loss of ATF3 reduces pancreatic cancer progression by limiting acinar-to-ductal metaplasia and enhancing regeneration, but KRASG12D can bypass ATF3's requirement in some contexts.

bioRxiv · Mar 27, 2020Cancer Biology