Author

C. L. Pin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Research Focus:cell type annotation· 4 Organism:mouse· 4 Assay:bulk RNA-seq· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:differential expression· 2 Organism:human· 2

Frequent Co-Authors

F. Mousavi· 2 shared M. Martin· 2 shared P. Shooshtari· 2 shared A. Cusimano· 1 shared A. El kaoutari· 1 shared A. Listi· 1 shared A. Mathison· 1 shared C. Johnson· 1 shared

Activity by Year

2020-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersCancer Biology

F. Mousavi

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

EZH2 deletion does not impact acinar cell regeneration but restricts progression to pancreatic cancer in mice

E. Jaune-Pons, X. Wang, F. Mousavi, A. El kaoutari, K. Berger, C. Johnson, M. Martin, S. Aggarwal, S. Brar, K. Muhammad, P. Shooshtari, A. Mathison, N. Dusetti, R. Urrutia, G. Lomberk, C. L. Pin

This study shows EZH2 deletion in adult acinar cells does not block regeneration but limits PDAC progression by altering the tumor microenvironment and differentiation.

bioRxiv · Sep 25, 2023 · PreprintCancer Biology

Defining the SPCA2C interactome identifies unique links to store-operated calcium entry

P. Samardzija, M. A. Fenech, R. Lang, M. C. Carter, S. Chen, S. Shi, P. B. Stathopulos, C. L. Pin

This study identifies novel SPCA2C interactors involved in calcium signaling and vesicular transport using BioID and validates key interactions with STIM1, ESYT1, and CCDC47.

bioRxiv · Sep 13, 2023 · PreprintCell Biology

Mouse models for pancreatic ductal adenocarcinoma are affected by the cre-driver used to promote KRASG12D activation.

F. Mousavi, J. Thompson, J. Lau, N. Renollet, M. B. Martin, J. McGue, T. Frankel, P. Shooshtari, C. L. Pin, F. Bednar

This study compares mouse models of pancreatic ductal adenocarcinoma driven by different cre-recombinase systems, finding differences in tumor development and inflammation.

bioRxiv · Sep 12, 2023 · PreprintCancer Biology

NUPR1 protects liver from lipotoxic injury by improving the endoplasmic reticulum stress response

M. T. Borrello, M. Emma, A. Listi, M. Rubis, S. Coslet, G. Augello, A. Cusimano, D. Cabibi, R. Porcasi, L. Giannitrapani, M. Soresi, G. Pantuso, K. Blyth, G. Montalto, C. L. Pin, , J. L. Iovanna

This study shows NUPR1 protects the liver from lipotoxic injury by enhancing the endoplasmic reticulum stress response and regulating lipid homeostasis through PPAR- signaling.

bioRxiv · Oct 23, 2020Cell Biology

Loss of Activating Transcription Factor 3 prevents KRAS-mediated pancreatic cancer

N. Azizi, J. Toma, M. Martin, M. F. Khalid, N. Steele, J. Shi, M. Pasca Di Magliano, C. L. Pin

This study shows that loss of ATF3 reduces pancreatic cancer progression by limiting acinar-to-ductal metaplasia and enhancing regeneration, but KRASG12D can bypass ATF3's requirement in some contexts.

bioRxiv · Mar 27, 2020Cancer Biology