Author

M. Ghazi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1

Frequent Co-Authors

A. Mishra· 1 shared A. Vats· 1 shared B. Prasher· 1 shared D. Singh· 1 shared F. Sultan· 1 shared I. Gupta· 1 shared K. Sharma· 1 shared M. Maulik· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

A. Mishra

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2 papers

Recent·Most cited

Sustained pigmentation causes DNA damage and invokes translesion polymerase Pol κ for repair in melanocytes

V. Natarajan, S. Khanna, M. Ghazi, Y. J. Subramaniam, I. Gupta, F. Sultan, K. Sharma, S. Chandna, R. S. Gokhale

This study shows sustained melanogenesis causes DNA damage in melanocytes, invokes Pol κ for repair, and links Pol κ activity to genome instability and mutational load in melanoma.

bioRxiv · May 20, 2022 · PreprintCell Biology

Baseline cell proliferation rates and response to UV differ in Lymphoblastoid Cell Lines derived from healthy individuals of extreme constitution types

S. Chakraborty, S. Singhmar, D. Singh, M. Maulik, R. Patil, S. K. Agrawal, A. Mishra, M. Ghazi, A. Vats, v. T. Natarajan, S. Juvekar, B. Prasher, M. Mukerji

The paper compares baseline cell proliferation rates and UV response in Lymphoblastoid Cell Lines from healthy individuals with different constitution types, finding significant differences in proliferation and recovery.

Cell Cycle · Apr 18, 2021Peer-reviewedCell Biology