Author

M. K. Jolly

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 51 Biophysics· 8 Cell Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Organism:human· 44 Approach:statistical model· 44 Assay:bulk RNA-seq· 32 Research Focus:cell type annotation· 27 Approach:experimental assay· 18 Research Focus:cell signaling & trafficking· 16 Research Focus:differential expression· 16 Research Focus:cancer & tumor microenvironment· 11

Frequent Co-Authors

H. Levine· 25 shared P. Chakraborty· 11 shared S. Sahoo· 10 shared K. Hari· 8 shared F. Bocci· 7 shared J. A. Somarelli· 7 shared J. N. Onuchic· 7 shared J. T. George· 7 shared

Activity by Year

2017-2023 · peak 14/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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25 shared papersCancer Biology

H. Levine

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Author profile →

62 papers

Recent·Most cited

Cancer BiologybioRxivMar 22, 2017

Inflammatory Breast Cancer: a model for investigating cluster-based dissemination

M. K. Jolly, M. Boareto, B. G. Debeb…·H. Levine

This review discusses molecular mechanisms enabling CTC clusters formation and their higher metastatic potential, using Inflammatory Breast Cancer as an example.

DOI 10.1038/s41523-017-0023-9

𝕏 PostBluesky

Cancer BiologybioRxivJan 6, 2017

Distinguishing Mechanisms Underlying EMT Tristability

D. Jia, M. K. Jolly, S. C. Tripathi…·H. Levine

The paper uses experimental and theoretical approaches to evaluate two models of EMT dynamics, finding experimental evidence supporting the ternary chimera switch model over the cascading bistable switches model.

DOI 10.1186/s41236-017-0005-8

humanexperimental assay

𝕏 PostBluesky