M. K. Madhu

This study uses molecular dynamics simulations to investigate how mutations in β2-adrenergic receptor cause biased signaling toward β-arrestin or G proteins.

This study uses molecular dynamics simulations to investigate phosphorylation-induced conformational changes in β2-adrenergic receptor, revealing a new state that facilitates β-arrestin binding.

This paper reconstructs a genome-scale metabolic model for Methylococcus capsulatus str. Bath and identifies key metabolic pathways and essential genes.