Author

M. Maio

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

C. Verollet· 3 shared D. Palmero· 3 shared F. Fuentes· 3 shared J. L. Marin Franco· 3 shared L. Balboa· 3 shared M. Genoula· 3 shared O. Neyrolles· 3 shared B. Dolotowicz· 2 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersImmunology

C. Verollet

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Elevated glycolytic metabolism of monocytes limits the generation of HIF-1α-driven migratory dendritic cells in tuberculosis

M. Maio, J. Barros, M. Joly, Z. Vahlas, J. L. Marin Franco, M. Genoula, S. Monard, M. B. Vecchione, F. Fuentes, V. Gonzalez Polo, M. F. Quiroga, M. Vermeulen, T. P. Vu Manh, R. J. Argüello, S. Inwentarz, , L. Balboa

This study shows that elevated glycolytic metabolism in monocytes during tuberculosis impairs their differentiation into migratory dendritic cells, limiting immune response.

bioRxiv · Apr 3, 2023Immunology

Landscape of immune-related signatures induced by targeting of different epigenetic regulators in melanoma: implications for immunotherapy

A. Anichini, A. Molla, G. Nicolini, V. E. Perotti, F. Sgambelluri, A. Covre, C. Fazio, M. F. Lofiego, A. M. Di Giacomo, S. Coral, A. Manca, M. C. Sini, M. Pisano, T. Noviello, F. Caruso, , R. Mortarini

This paper identifies immune-related gene signatures induced by four epigenetic inhibitors in melanoma cells, finding that guadecitabine most strongly upregulates immune-related genes and activates innate immunity pathways.

bioRxiv · Apr 13, 2022Immunology

Host-derived lipids from tuberculous pleurisy impair macrophage microbicidal-associated metabolic activity

J. L. Marin Franco, M. Genoula, D. Corral, G. Duette, M. Ferreyra, M. Maio, B. Dolotowicz, O. E. Aparicio-Trejo, E. Patino-Martinez, F. Fuentes, V. Soldan, E. J. Morana, D. Palmero, M. Ostrowski, P. Schierloh, , L. Balboa

This study shows that host-derived lipids from tuberculous pleurisy reduce glycolytic activity and impair HIF-1-mediated metabolic reprogramming in macrophages, promoting Mtb infection.

bioRxiv · Mar 23, 2020Immunology

Mycobacterium tuberculosis Modulates the Metabolism of Alternatively Activated Macrophages to Promote Foam Cell Formation and Intracellular Survival

M. Genoula, J. L. Marin Franco, M. Maio, B. Dolotowicz, M. Ferreyra, M. A. Milillo, R. Mascarau, E. J. Morana, D. Palmero, F. Fuentes, B. Lopez, P. Barrionuevo, O. Neyrolles, C. Cougoule, G. Lugo-Villarino, , L. Balboa

This study shows that Mtb modulates macrophage metabolism to promote foam cell formation and survival by altering lipid metabolism and FAO pathways.

bioRxiv · Dec 13, 2019Immunology