Author

M. Mayo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:experimental assay· 2 Research Focus:phylogenetics / evolution· 2 Approach:statistical model· 2 Assay:whole-genome sequencing· 2 Organism:bacteria (other)· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

B. J. Currie· 4 shared D. S. Sarovich· 3 shared E. P. Price· 3 shared J. R. Webb· 2 shared A. Aziz· 1 shared A. J. Vazquez· 1 shared A. S. Amiss· 1 shared A. Tuanyok· 1 shared

Activity by Year

2017-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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4 shared papersMicrobiology

B. J. Currie

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Author profile →

4 papers

Recent·Most cited

Drug susceptibility profiling of Australian Burkholderia species as models for developing melioidosis therapeutics

A. S. Amiss, J. R. Webb, M. Mayo, B. J. Currie, D. J. Craik, S. Troeira Henriques, N. Lawrence

The paper profiles drug susceptibility of Australian Burkholderia species to identify models for developing melioidosis therapeutics, finding high similarity in susceptibility profiles between B. pseudomallei and less pathogenic species.

The American Journal of Tropical Medicine and Hygiene · Nov 4, 2020Peer-reviewedMicrobiology

Comparative genomics confirms a rare melioidosis human-to-human transmission event and reveals incorrect phylogenomic reconstruction due to polyclonality

A. Aziz, B. J. Currie, M. Mayo, D. S. Sarovich, E. P. Price

This paper investigates a rare human-to-human transmission of Burkholderia pseudomallei from mother to child using whole-genome sequencing, revealing polyclonality in the mother and demonstrating how mixture can mislead phylogenomic analysis.

bioRxiv · Oct 15, 2019Microbiology

Pathogen to commensal: longitudinal within-host population dynamics, evolution, and adaptation during a chronic >16-year Burkholderia pseudomallei infection

T. Pearson, J. Sahl, C. M. Hepp, K. Handady, H. Hornstra, A. J. Vazquez, E. Settles, M. Mayo, M. Kaestli, C. H. D. Williamson, E. P. Price, D. S. Sarovich, J. M. Cook, S. R. Wolken, R. A. Bowen, , P. Keim

This study tracks the long-term evolution of Burkholderia pseudomallei in a human host over 16 years, showing its transition from a pathogen to a commensal through genetic and phenotypic changes.

bioRxiv · Feb 17, 2019Evolutionary Biology

Raising the stakes: Loss of efflux-pump regulation decreases meropenem susceptibility in Burkholderia pseudomallei

D. S. Sarovich, J. R. Webb, M. C. Pitman, L. T. Viberg, M. Mayo, R. W. Baird, J. M. Robson, B. J. Currie, E. P. Price

This study identifies mutations in RND efflux pump regulators in B. pseudomallei isolates that lead to decreased meropenem susceptibility and are associated with fatal melioidosis outcomes.

bioRxiv · Oct 20, 2017Microbiology