M. Schwarzländer

The paper shows that UCP2 in Arabidopsis localizes to the Golgi instead of mitochondria, challenging previous assumptions about its function and highlighting the need for independent localization evidence.

The paper generates and characterizes barley plants expressing Grx1-roGFP2, showing cytosolic glutathione redox potential remains stable under combined salt and osmotic stress.

The paper investigates how photosynthetic activity affects subcellular pH and NAD redox status in Arabidopsis cells using fluorescent biosensors.

The paper identifies lysine acetylation as a conserved regulatory mechanism in land plants, showing it fine-tunes mitochondrial malate dehydrogenase activity in a species-specific manner.

The paper uses redox biosensors to show that chloroplast-derived ROS from methyl viologen causes H2O2 and glutathione oxidation in chloroplasts, cytosol, and mitochondria.

This paper identifies DIC2 as a bifunctional mitochondrial carrier in Arabidopsis that facilitates malate import and citrate export, maintaining metabolic homeostasis in leaves.

The paper shows that GRXS15 deficiency disrupts mitochondrial lipoyl cofactor homeostasis, leading to metabolic defects in lipoyl cofactor-dependent dehydrogenase complexes, particularly affecting branched-chain amino acid catabolism.

The paper phenotypically characterizes low-glutathione Arabidopsis mutants under control and drought conditions, finding reduced growth but no increased drought sensitivity.

The paper investigates the role of GR2 in maintaining glutathione redox homeostasis in plastids and mitochondria of Arabidopsis, showing its essential function in plastids and partial redundancy in mitochondria.

This study shows that glutathione reductase is essential for maintaining a reducing stromal glutathione redox potential in chloroplasts, which supports efficient photosynthesis and plant growth.