Author

Matthew Diaz

Harvard University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Alejandro Sweet-CorderoiD· 1 shared Catherine RheeiD· 1 shared David B. SykesiD· 1 shared David T. ScaddeniD· 1 shared J. Alex B. Gibbons· 1 shared Jacob D. BhoiiD· 1 shared Jennifer A. EvansiD· 1 shared Jonathan G. Van VrankeniD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCancer Biology

Alejandro Sweet-CorderoORCID

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2 papers

Recent·Most cited

Melanopsin contributes to circadian photic responses in mice in a sex-dependent manner

Kayla C. Miguel, Marcos L. Aranda, Jacob D. Bhoi, Matthew Diaz, Jennifer A. Evans, Tiffany M. Schmidt

This study shows that melanopsin expression and its role in circadian photic responses differ between male and female mice.

bioRxiv · Dec 13, 2025 · PreprintNeuroscience

Interrupting ELMSAN1 repression of nuclear Acetyl-CoA production therapeutically reprograms cancer cells

Ting Zhao, Lingli He, Lai Ping Wong, Shenglin Mei, Jun Xia, Yanxin Xu, Jonathan G. Van Vranken, Michael C. Mazzola, Lei Chen, Catherine Rhee, Tiancheng Fang, Tsuyoshi Fukushima, Leanne C. Sayles, Matthew Diaz, J. Alex B. Gibbons, , David T. Scadden

The paper identifies ELMSAN1 as a repressor of nuclear acetyl-CoA production and shows that inhibiting this interaction reprograms cancer cells to a postmitotic state, improving survival in multiple cancer models.

bioRxiv · Apr 25, 2025 · PreprintCancer Biology