Author

P. S. Ginter

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 2 Organism:human· 2 Assay:organoid / primary culture· 2 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

B. Awuah· 2 shared B. Stonaker· 2 shared E. B. Osei-Bonsu· 2 shared E. Cheng· 2 shared E. K. Adjei· 2 shared F. S. Aitpillah· 2 shared I. Kyei· 2 shared J. K. Oppong· 2 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCancer Biology

B. Awuah

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2 papers

Recent·Most cited

Cancer BiologyCancer ResearchPeer-reviewedFeb 1, 2025

Kinome focused CRISPR-Cas9 screens in African ancestry patient-derived breast cancer organoids identifies essential kinases and synergy of EGFR and FGFR1 inhibition.

F. P. Madorsky Rowdo, R. Martini, S. E. Ackermann…·M. L. Martin

The paper uses CRISPR-Cas9 screens in breast cancer organoids from African ancestry patients to identify essential kinases and show synergy between EGFR and FGFR1 inhibitors.

DOI 10.1158/0008-5472.can-24-0775

humanorganoid / primary culture

𝕏 PostBluesky

Cancer BiologybioRxivJun 22, 2023

Patient-derived tumor organoids with p53 mutations, and not wild-type p53, are sensitive to synergistic combination PARP inhibitor treatment

F. P. M. Rowdo, G. Xiao, G. F. Khramtsova…·J. Bargonetti

This study shows that tumor organoids with mutant p53 are more sensitive to the combination of PARP inhibitor and temozolomide than those with wild-type p53.

DOI 10.1016/j.canlet.2024.216608

humanorganoid / primary culture

𝕏 PostBluesky