Author

R. Bechara

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

S. Gaffen· 2 shared C. Jawale· 1 shared C. Zhou· 1 shared D. H. Kaplan· 1 shared D.-D. Li· 1 shared F. E. Aggor· 1 shared J. Kolls· 1 shared K. Ramani· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersImmunology

S. Gaffen

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2 papers

Recent·Most cited

Antibody-induced glomerulonephritis is amplified by RTEC-intrinsic IL-17 signaling and restrained by IL-17-mediated induction of the endoribonuclease Regnase-1 (Zc3h12a)

D.-D. Li, R. Bechara, K. Ramani, C. Jawale, Y. Li, J. Kolls, S. Gaffen, P. S. Biswas

This study shows that IL-17 signaling in renal tubular epithelial cells drives antibody-induced glomerulonephritis, and Regnase-1 limits this inflammation by inhibiting IL-17 target genes.

bioRxiv · Jan 11, 2021 · PreprintImmunology

A loss-of-function mutation in IL-17F enhances susceptibility of mice to oropharyngeal candidiasis

C. Zhou, L. Monin, R. Gordon, F. E. Aggor, R. Bechara, T. Edwards, D. H. Kaplan, S. Gingras, s. gaffen

This study shows that a loss-of-function mutation in IL-17F increases susceptibility to oropharyngeal candidiasis in mice, but not in heterozygous carriers.

bioRxiv · Apr 9, 2020Immunology