Author

S. Bayraktar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Organism:Drosophila· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Janning· 1 shared C. Schuberth· 1 shared E. Menis· 1 shared H. Pavenstädt· 1 shared J. A. Cyr· 1 shared J. Halbritter· 1 shared J. Nehrig· 1 shared J. Stebbing· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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1 shared paperCell Biology

A. Janning

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cell BiologybioRxivPreprintDec 6, 2023

CRISPR screens in 3D tumourspheres identified miR-4787-3p as a transcriptional start site miRNA essential for breast tumour-initiating cell growth

T. Stiff, S. Bayraktar, P. Dama…·L. Castellano

The paper identifies miR-4787-3p as an essential miRNA for breast tumor-initiating cell growth using CRISPR screens and validates its role as a potential therapeutic target and prognostic biomarker.

DOI 10.1101/2023.12.06.570391

humanbulk RNA-seq

𝕏 PostBluesky

BioengineeringbioRxivAug 6, 2021

Unravelling Form and Function: Improved function of engineered cardiac tissue through extra-cellular anisotropy

J. A. Cyr, M. Colzani, S. Bayraktar…·R. E. Cameron

This study shows that aligned collagen scaffolds improve cardiac tissue function by enhancing signaling synchronicity and contractility compared to isotropic scaffolds.

DOI 10.1016/j.bioadv.2023.213680

humanexperimental assay

𝕏 PostBluesky

Cell BiologybioRxivNov 12, 2019

A deregulated stress response underlies distinct INF2 associated disease profiles

S. Bayraktar, J. Nehrig, E. Menis…·R. Wedlich-Soldner

The paper identifies distinct disease profiles caused by INF2 mutations through cellular profiling, showing varying stress responses and linking them to FSGS and CMT.

DOI 10.1681/asn.2019111174

Drosophilaproteomics

𝕏 PostBluesky