Author

S. W. Wong

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:drug response / drug discovery· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Assay:CRISPR screen· 1 Approach:deep learning· 1 Approach:statistical model· 1

Frequent Co-Authors

A. P. Wiita· 3 shared J. L. Wolf· 3 shared N. Shah· 3 shared P. Choudhry· 3 shared T. G. Martin· 3 shared I. D. Ferguson· 2 shared Y.-H. T. Lin· 2 shared A. B. Abarientos· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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3 shared papersCancer Biology

A. P. Wiita

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3 papers

Recent·Most cited

Cancer BiologyNature CommunicationsPeer-reviewedJul 15, 2022

Defining the cell surface proteomic landscape of multiple myeloma reveals immunotherapeutic strategies and biomarkers of drug resistance

I. D. Ferguson, B. P. Escobar, S. T. Tuomivaara…·A. P. Wiita

This study defines the myeloma cell surface proteome, identifies therapeutic targets and resistance biomarkers, and presents a miniaturized surface proteomics protocol.

DOI 10.1038/s41467-022-31810-6

humanproteomics

𝕏 PostBluesky

Cancer BiologybioRxivApr 14, 2020

CRISPR-based screens uncover determinants of immunotherapy response in Multiple Myeloma

P. Ramkumar, A. B. Abarientos, R. Tian…·M. Kampmann

This study uses CRISPR screens to identify mechanisms increasing BCMA expression and enhancing anti-myeloma efficacy of BCMA-targeted therapies in multiple myeloma.

DOI 10.1182/bloodadvances.2019001346

humanCRISPR screen

𝕏 PostBluesky

Cancer BiologybioRxivFeb 3, 2019

Proteasome inhibitor-induced modulation reveals the spliceosome as a specific therapeutic vulnerability in multiple myeloma

H. H.-L. Huang, I. D. Ferguson, A. M. Thornton…·A. P. Wiita

The paper shows proteasome inhibitors modulate splicing in multiple myeloma, revealing spliceosome inhibition as a therapeutic target.

DOI 10.1038/s41467-020-15521-4

humanproteomics

𝕏 PostBluesky