Author

Sammy Alhassen

University of California, Irvine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:metabolomics· 2 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Amal AlachkariD· 2 shared Alvin PhaniD· 1 shared Angele De SilvaiD· 1 shared Archana ProdduturiD· 1 shared Carlos Portera-Cailliau· 1 shared Charity Henrich· 1 shared Chelsea Marie T. ParrochaiD· 1 shared Dana Shevachman· 1 shared

Activity by Year

2021-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersNeuroscience

Amal AlachkarORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintNov 11, 2026

A protocol for cortical and deep-brain calcium imaging in freely moving mice with the Mini2P system.

Irma A. Tello Garcia, Sammy Alhassen, Carlos Portera-Cailliau, William Zeiger

This paper provides a detailed protocol for implementing Mini2P calcium imaging in freely moving mice, covering system setup, optical alignment, and behavioral integration to study neural activity during natural behavior.

DOI 10.64898/2026.11.09.764980

mouseimaging

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NeurosciencebioRxivPreprintAug 4, 2026

Primary Cilia Loss in Striatal Neurons Disrupts Synaptic Connectivity and Excitatory Transmission and Drives Metabolomic Remodeling

Kiki Chen, Archana Proddutur, Dana Shevachman…·Amal Alachkar

DOI 10.64898/2026.07.30.741876

mousemetabolomics

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NeuroscienceCommunications BiologyPeer-reviewedJun 24, 2021

Intergenerational Stress Transmission is Associated with Brain Metabotranscriptome Remodeling andMitochondrial Dysfunction

Sammy Alhassen, Siwei Chen, Lamees Alhassen…·Amal Alachkar

This study shows that prenatal stress exposure in mice leads to behavioral deficits in offspring, associated with mitochondrial dysfunction and epigenetic changes, which can be mitigated by acetyl-L-carnitine.

32 citationstop 4%

mouseproteomics

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