Author

T. Eagar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:flow cytometry· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

A. Cross· 1 shared B. Edelson· 1 shared G. Wu· 1 shared J. Herz· 1 shared L. Calvier· 1 shared N. Loof· 1 shared N. Manouchehri· 1 shared O. Stuve· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

A. Cross

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

CD11c+CD88+CD317+ myeloid cells are critical mediators of persistent CNS autoimmunity

N. Manouchehri, R. Hussain, P. Cravens, B. Edelson, G. Wu, A. Cross, R. Doelger, N. Loof, T. Eagar, T. Forsthuber, L. Calvier, J. Herz, O. Stuve

This study identifies CD11c+CD88+CD317+ myeloid cells as key drivers of CNS autoimmunity and shows that targeting these cells with anti-CD317 mAb reduces disease.

bioRxiv · Jun 10, 2020Immunology