Author

B. Edelson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Approach:experimental assay· 1 Assay:flow cytometry· 1

Frequent Co-Authors

A. Cross· 1 shared G. Wu· 1 shared G.-R. Kim· 1 shared H.-G. Lee· 1 shared J. Herz· 1 shared J.-H. Koo· 1 shared J.-M. Choi· 1 shared J.-W. Yoon· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

A. Cross

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Bystander memory-phenotype conventional CD4+ T cells exacerbating autoimmune neuroinflammation

M.-Z. Cho, H.-G. Lee, J.-W. Yoon, G.-R. Kim, J.-H. Koo, R. Taneja, B. Edelson, Y. J. Lee, J.-M. Choi

The paper identifies bystander memory-phenotype CD4+ T cells that contribute to autoimmune neuroinflammation through TCR-independent effector functions and GM-CSF production.

bioRxiv · Jun 17, 2022 · PreprintImmunology

CD11c+CD88+CD317+ myeloid cells are critical mediators of persistent CNS autoimmunity

N. Manouchehri, R. Hussain, P. Cravens, B. Edelson, G. Wu, A. Cross, R. Doelger, N. Loof, T. Eagar, T. Forsthuber, L. Calvier, J. Herz, O. Stuve

This study identifies CD11c+CD88+CD317+ myeloid cells as key drivers of CNS autoimmunity and shows that targeting these cells with anti-CD317 mAb reduces disease.

bioRxiv · Jun 10, 2020Immunology