Author

T. Eder

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3 Microbiology· 1 Scientific Communication & Education· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 4 Assay:bulk RNA-seq· 3 Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:chromatin & epigenetic regulation· 2 Approach:statistical model· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

F. Grebien· 3 shared E. Heyes· 2 shared T. Haferlach· 2 shared A. Boersma· 1 shared A. C. Müller· 1 shared A. Duchowski· 1 shared A. Pichler· 1 shared A. S. Wilhelmson· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersCancer Biology

F. Grebien

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

TET2 lesions enhance the aggressiveness of CEBPA-mutant AML by rebalancing GATA2 expression

E. Heyes, A. S. Wilhelmson, A. Wenzel, G. Manhart, T. Eder, M. B. Schuster, E. Rzepa, S. Pundhir, T. D'Altri, C. Gentil, M. Meggendorfer, J. Schwaller, T. Haferlach, F. Grebien, B. T. Porse

This study identifies GATA2 as a target of the CEBPA-TET2 mutational axis in AML, showing how TET2 lesions rebalance GATA2 expression and enhance disease aggressiveness.

bioRxiv · Mar 28, 2023Cancer Biology

STAT5 Gain-of-Function Variants Promote Precursor T-Cell Receptor Activation to Drive T-Cell Acute Lymphoblastic Leukemia

T. Suske, H. Sorger, F. Ruge, N. Prutsch, M. W. Zimmerman, T. Eder, B. Maurer, C. Wagner, S. Schoenefeldt, K. Spirk, A. Pichler, T. Pemovska, C. Schweicker, D. Poeloeske, D. Jungherz, , R. Moriggl

This study identifies STAT5 gain-of-function mutations as drivers of T-ALL by promoting pre-TCR signaling and shows therapeutic targeting potential with STAT3/5 or ZAP70 inhibitors.

bioRxiv · Dec 21, 2022 · PreprintCancer Biology

Antiviral immune response reveals host-specific virus infections in natural ant populations

L. Viljakainen, M. A. Fuerst, A. V. Grasse, J. Jurvansuu, J. Oh, L. Tolonen, T. Eder, T. Rattei, S. Cremer

The paper identifies host-specific viruses in ant populations using RNA-seq and sRNA-seq, revealing high host-specificity of active infections and spillover of non-infecting contaminants.

bioRxiv · Oct 17, 2022 · PreprintMicrobiology

Biomolecular Condensation Drives Leukemia Caused by NUP98-Fusion Proteins

S. Terlecki-Zaniewicz, T. Eder, J. Schmoellerl, T. Humer, N. Kuchynka, K. Parapatics, E. Heyes, F. G. Liberante, A. C. Müller, F. Grebien

This study identifies biomolecular condensation as a mechanism by which NUP98-fusion proteins drive leukemia by altering condensate composition and gene regulation.

bioRxiv · Nov 16, 2020 · PreprintCancer Biology

Pupil diameter differentiates expertise in dental radiography visual search

N. Castner, T. Appel, T. Eder, J. Ricther, K. Scheiter, C. Keutel, F. Hüttig, A. Duchowski, E. Kasneci

This study compares pupil diameter responses to varying difficulty anomalies in dental radiographs between expert and student dentists, finding experts show proportional cognitive load responses while students show consistently higher load.

bioRxiv · Oct 3, 2019Scientific Communication & Education