Torri D. Wilson

This study identifies CGRPR-expressing neurons in the central amygdala as key players in pain hypersensitivity, showing their role in both injury-induced and spontaneous pain responses.

This study shows the PbN[->]CeA circuit contributes to injury-induced pain hypersensitivity but not baseline nociception in mice.

This study identifies an inhibitory neural circuit from CeA-PKCδ neurons to the zona incerta that bidirectionally modulates pain-related behaviors in mice.

This study identifies 4R-Tobacco-Cembranoid as a positive allosteric modulator of 7nAChRs that reduces thermal hypersensitivity and inflammation in mice, with sex-specific effects.

This study examines sex differences in visceral pain behaviors and disease progression in mouse models of acute and persistent colon inflammation, finding sex-specific responses and disease outcomes.

This study identifies cell-type-specific differences in excitability and morphology between PKCδ+ and Som+ neurons in the central amygdala and links these differences to behavioral changes in neuropathic pain.