Author

Y. Hirayama

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2

Frequent Co-Authors

J. Gohda· 2 shared J.-i. Inoue· 2 shared K. Tomita· 2 shared M. Yamamoto· 2 shared Y. Kawaguchi· 2 shared A. Kobayashi· 1 shared K. Semba· 1 shared M. Seiki· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersMicrobiology

J. Gohda

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2 papers

Recent·Most cited

MicrobiologybioRxivPreprintMar 21, 2022

SARS-CoV-2 Omicron spike H655Y mutation is responsible for enhancement of the endosomal entry pathway and reduction of cell surface entry pathway

M. Yamamoto, K. Tomita, Y. Hirayama…·J. Gohda

This paper investigates how the H655Y mutation in the Omicron SARS-CoV-2 spike protein enhances endosomal entry and reduces cell surface entry, altering viral tropism and fusogenicity.

DOI 10.1101/2022.03.21.485084

humanexperimental assay

𝕏 PostBluesky

MicrobiologybioRxivPreprintDec 14, 2021

Metalloproteinase-dependent and TMPRSS2-independnt cell surface entry pathway of SARS-CoV-2 requires the furin-cleavage site and the S2 domain of spike protein

M. Yamamoto, J. Gohda, A. Kobayashi…·J.-i. Inoue

This study identifies a TMPRSS2-independent metalloproteinase-dependent entry pathway for SARS-CoV-2 that requires the furin cleavage site and S2 domain of the spike protein, highlighting new therapeutic targets.

DOI 10.1101/2021.12.14.472513

humanexperimental assay

𝕏 PostBluesky