Bartłomiej Waclaw

The paper tests a birth-death-mutation model's ability to predict antibiotic resistance evolution in experiments, finding it partially successful with model modifications improving predictions.

The paper quantifies phenotypic heterogeneity in glioblastoma cells' response to temozolomide, showing non-genetic factors drive differential drug response.

This study uses a mathematical model to show that non-planktonic bacterial phases in UTIs increase infection risk and shape resistance evolution, with probiotic pretreatment improving antibiotic efficacy.

The paper develops a roadblock-and-kill model to explain how E. coli responds dynamically to ciprofloxacin, showing that DNA replication blockage, not the SOS response, drives growth inhibition.