Angela J Dawson

This study investigates how E. coli bacteria navigate complex non-Newtonian fluids, finding that mechanical properties of the medium override their natural motility strategies.

This study shows low-dose antibiotics trigger bacterial aggregation via lysis and extracellular DNA release, forming aggregates distinct from biofilms.

The paper develops a roadblock-and-kill model to explain how E. coli responds dynamically to ciprofloxacin, showing that DNA replication blockage, not the SOS response, drives growth inhibition.