Maria-Halima Laaberki

This paper identifies calcium as a key inducer of natural transformation in Acinetobacter baumannii, revealing both transcriptional and structural mechanisms by which calcium activates type IV pilus function.

This study examines genetic and phenotypic changes in Acinetobacter baumannii during a five-year UTI without antibiotic treatment, revealing genome reduction and loss of adhesion capacity but conserved colistin resistance.

This study investigates how the Xer recombination system in Acinetobacter baumannii facilitates plasmid co-integration and gene exchange, contributing to the spread of antibiotic resistance.

This study shows AbaR-type islands persist in Acinetobacter baumannii by integrating into comM, reducing their removal while allowing continued genetic exchange.