Kim Tieu

This study identifies DRP1 as a transcription factor that regulates neuroinflammation by activating NF-κB and lipocalin 2, offering potential therapeutic targets for neurodegenerative diseases.

This study shows increased DRP1/Drp1 and altered fission/fusion proteins in human and mouse Parkinson's brains, suggesting targeting mitochondrial fission could be a therapeutic strategy.

This study shows that partial Drp1 knockout enhances autophagy flux without affecting mitochondrial function, revealing a distinct protective mechanism against manganese toxicity.