Gabriele Chelini

This study shows that oxidative stress and inflammation interact to support autistic-like behaviors in Cntnap2-deficient mice, with NAC treatment improving these behaviors and microglia function.

This paper uses automated body-language analysis to show that a dual-hit mouse model of psychiatric vulnerability exhibits distinct neurobehavioral profiles shaped by genotype and early-life adversity.

This study shows that N-acetylcysteine reduces inflammation and improves social behavior in a Shank3b mouse model of ASD.

This study shows that early-life adversity in Cntnap2 heterozygous mice leads to bipolar-like behaviors and synaptic changes in the amygdala.

The paper analyzes single-nucleus RNA sequencing data from marmoset brain cells to show that neuronal identity is more influenced by developmental origin than functional class.

This study investigates somatosensory processing deficits in Shank3b-/- mice, finding reduced cortical and hippocampal activation and connectivity linked to hypo-responsiveness to tactile stimuli.

The paper identifies CS6 clusters as extracellular matrix microenvironments that support coordinated multi-dendrite synaptic plasticity in response to sensory stimuli.

The paper investigates circadian rhythms of perineuronal nets (PNNs) in rodents and humans, showing diurnal and circadian variations and disruption by sleep deprivation.