Olivier Hamant

This paper develops a system to apply quantifiable mechanical stress to plant cells and observe their responses using confocal imaging, aiming to uncover molecular mechanisms of touch response.

This study identifies TCP4 as a key regulator of leaf shape by integrating genetic and mechanical signals across tissue layers in Arabidopsis.

The paper investigates how cytoskeleton, nucleus dynamics, and cell mechanics coordinate root hair growth phases in Arabidopsis using live imaging and mathematical modeling.

The paper shows MAP65-1/PRC1 promotes microtubule nucleation on damaged lattices under mechanical stress, stabilizing microtubules and reinforcing their alignment with stress.

This paper identifies the lectin domain of LecRK-I.9 as responsible for creating persistent plasma membrane-cell wall connections in plants under water stress.

The paper shows plant protoplasts can form protrusions, challenging the view of plant cells as static and revealing similarities with animal cells.

This study identifies CUC2 as a direct activator of MIR319C transcription, which delays the cell proliferation-to-differentiation transition in Arabidopsis leaf primordia, leading to smaller leaves.

The paper identifies a 3-component module involving VIP4, ARF3, and VANGUARD1 that maintains sepal flatness in Arabidopsis by regulating cell wall stiffness and growth balance.

The paper quantitatively investigates how cell size variation affects sepal shape robustness in Arabidopsis, finding that cell size heterogeneity is not essential for shape robustness.

The paper shows that Paf1C reduces transcriptional noise in Arabidopsis, leading to more consistent organ shape by managing mechanical conflicts.

The paper shows that cell lineages in Arabidopsis sepal develop from size uniformization to enhancement of size variability during growth.