Maria Eugenia Teves

This study identifies AURKA/HDAC6 activation as a driver of primary cilia shortening and fibroblast activation in systemic sclerosis.

This study identifies ciliary program imbalance as a driver of fibroblast-to-myofibroblast reprogramming in systemic sclerosis, suggesting ciliotherapy as a potential treatment.

The paper shows that altering primary cilia length in fibroblasts can reduce fibrotic responses across different fibrotic conditions.

The paper shows that species-specific PRM1 cysteine residues determine nuclear shape and histone retention in sperm by analyzing mutant mice.

This study identifies Spag17 as a key modulator of female reproductive aging, showing its role in maintaining fertility and preventing age-related complications.