Winnie Zhao

The paper shows that certain bacteria reduce microsporidia infection in C. elegans by altering host metabolism and producing inhibitory compounds.

This study identifies drug targets and resistance mechanisms in microsporidia using forward genetics in C. elegans.

This study examines how protein reduction in microsporidia affects function, showing that most proteins cannot complement yeast orthologs and are more reduced in length.

This study shows that parental vitamin B12 exposure in C. elegans leads to intergenerational growth acceleration and pathogen tolerance in offspring.

The paper systematically analyzes all 19 AGO proteins in C. elegans to uncover gene regulatory networks and novel phenotypes associated with their loss.

The paper identifies a C. elegans strain with opposing resistance and susceptibility to different microsporidia species through phenotyping and genetic analysis.