Author

Shuchi Agrawal Singh

University of CambridgeORCID 0000-0001-8632-4556Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:benchmarking· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Brian J.P. HuntlyiD· 2 shared Dhoyazan AzaziiD· 2 shared George GiotopoulosiD· 2 shared Jaana BagriiD· 2 shared Ryan J. AsbyiD· 2 shared Sarah J. HortoniD· 2 shared Albert KoulmaniD· 1 shared Alicia Garcia-GimeneziD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

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2 shared papersCancer Biology

Brian J.P. HuntlyORCID

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2 papers

Recent·Most cited

Cancer BiologyNature CommunicationsPeer-reviewedMay 20, 2025

Genetic or pharmacological inactivation of CREBBP sensitizes B-cell Acute Lymphoblastic Leukemia to Ferroptotic Cell Death upon BCL2 Inhibition

Alicia Garcia-Gimenez, Jonathan E. Ditcham, Dhoyazan Azazi…·Simon E. Richardson

This study identifies CREBBP mutations as a targetable vulnerability in B-ALL, showing that CREBBP inhibition sensitizes these cells to Venetoclax-induced ferroptosis.

19 citationstop 1%

humanbulk RNA-seq

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Molecular BiologybioRxivPreprintJan 27, 2025

Delayed Trp53 activation protects Dnmt3a-mutant hematopoietic stem cells from inflammatory attrition.

Ludovica Marando, George Giotopoulos, Pedro Madrigal…·Brian J.P. Huntly

This study shows that DNMT3A R882H mutant HSCs resist inflammatory stress through delayed p53 activation, leading to clonal expansion and increased AML risk.

DOI 10.1101/2025.01.27.635076

mousebulk RNA-seq

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