Author

Massimo Vetralla

University of PaduaORCID 0000-0001-8632-7634Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Organism:C. elegans· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1

Frequent Co-Authors

Asrat Kahsay· 1 shared Beijia Xie· 1 shared Dan EhningeriD· 1 shared Daniele BanoiD· 1 shared Diego De StefaniiD· 1 shared Enzo ScifoiD· 1 shared Lena WischhofiD· 1 shared Maëlle Sandhira HabertiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperBiochemistry

Asrat Kahsay

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1 paper

Recent·Most cited

BiochemistryNature CommunicationsPeer-reviewedDec 17, 2025

TMEM65-dependent Ca2+ extrusion safeguards mitochondrial homeostasis

Massimo Vetralla, Lena Wischhof, Asrat Kahsay…·Diego De Stefani

This study identifies TMEM65 as a key component of the mitochondrial Ca2+ efflux machinery, showing its role in maintaining Ca2+ homeostasis and preventing mitochondrial dysfunction.

33 citationstop 2%

C. elegansexperimental assay

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